CEREBRAL TOXOCARIASIS AFTER CONSUMPTION OF RAW DUCK LIVER

BODO HOFFMEISTER Department of Infectious Diseases and Pulmonology, Charité University Hospital, Berlin, Germany; Department of Neurology, Schlosspark-Klinik, Berlin, Germany; Department of Infectious Diseases and Pulmonology, Charité University Hospital, Berlin, Germany

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SVEN GLAESER Department of Infectious Diseases and Pulmonology, Charité University Hospital, Berlin, Germany; Department of Neurology, Schlosspark-Klinik, Berlin, Germany; Department of Infectious Diseases and Pulmonology, Charité University Hospital, Berlin, Germany

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HOLGER FLICK Department of Infectious Diseases and Pulmonology, Charité University Hospital, Berlin, Germany; Department of Neurology, Schlosspark-Klinik, Berlin, Germany; Department of Infectious Diseases and Pulmonology, Charité University Hospital, Berlin, Germany

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SEBASTIAN PORNSCHLEGEL Department of Infectious Diseases and Pulmonology, Charité University Hospital, Berlin, Germany; Department of Neurology, Schlosspark-Klinik, Berlin, Germany; Department of Infectious Diseases and Pulmonology, Charité University Hospital, Berlin, Germany

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NORBERT SUTTORP Department of Infectious Diseases and Pulmonology, Charité University Hospital, Berlin, Germany; Department of Neurology, Schlosspark-Klinik, Berlin, Germany; Department of Infectious Diseases and Pulmonology, Charité University Hospital, Berlin, Germany

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FRANK BERGMANN Department of Infectious Diseases and Pulmonology, Charité University Hospital, Berlin, Germany; Department of Neurology, Schlosspark-Klinik, Berlin, Germany; Department of Infectious Diseases and Pulmonology, Charité University Hospital, Berlin, Germany

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Human toxocariasis is usually contracted by exposure to contaminated soil. This disease is rarely transmitted by raw meat or giblets of paratenic animals, such as chickens, lambs, or cows. We present a case of isolated cerebral toxocariasis presumably caused by the consumption of raw duck liver. This 55-year-old woman had sudden-onset hemiparesis of the right leg, eosinophilia of 30%, and markedly elevated total serum IgE levels. Magnetic resonance imaging demonstrated multiple cerebral hyperintense lesions on T2-weighed images. Tests for antibodies to Toxocara in serum and cerebrospinal fluid yielded highly positive results. Repeated courses of albendazole and corticosteroids led to significant clinical improvement.

Author Notes

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