1921
Volume 27, Issue 5
  • ISSN: 0002-9637
  • E-ISSN: 1476-1645
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Abstract

Abstract

In the past, investigators have been able to produce hepatic amebiasis in laboratory animals only by direct introduction of parasites into the liver or its vasculature, or by other artificial manipulations. A natural model of human visceral amebiasis has been lacking. We document an extensive outbreak of amebic dysentery which took place in a colony of spider monkeys; severe hepatic abscesses occurred in many animals. The spider monkey is highly susceptible to infection with and could provide a valuable model for the study of the pathogenesis of invasive amebiasis.

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/content/journals/10.4269/ajtmh.1978.27.888
1978-09-01
2017-11-20
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http://instance.metastore.ingenta.com/content/journals/10.4269/ajtmh.1978.27.888
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  • Accepted : 28 Jan 1978

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